A plain-language guide

G6PD deficiency

What is known, what is still uncertain, and what is actively debated, written plainly, and built only from published medical research.

Early map · 13 sourced statements Every statement names its source Updated 2026-06-12
Please read this first. This guide is a companion to your medical team, not a replacement, and it is not medical advice. Everything here is tied to published research. If something you expected is not here, it almost always means we have not mapped a source for it yet, not that it is unknown to medicine. G6PD deficiency is an early, growing map, so it will look incomplete on purpose: we would rather show less and have every line be something you can check than fill the page with claims we cannot stand behind. For anything about your own situation, your clinicians hold the full picture. How this guide is built and why.

What it is

G6PD deficiency is an inherited difference in red blood cells in which oxidative stressors, such as certain drugs or eating fava beans, can trigger sudden episodes of red-cell breakdown (acute hemolysis).

Signs and symptoms

Hemoglobinuria

Hemoglobinuria means the urine turns dark because pigment from broken-down red blood cells passes into it. It can appear during an episode of red-cell breakdown.

Limited evidenceSource: OMIM:300908
Evidence ratingweak
Study designontology_import
Confidence (0-1)0.7
Replicationunreplicated
Supporting sourcesPMID:42017087
Notesplain_language confirmed from PMID:42017087 via curation 2026-06-12.
Last reviewed2026-06-12

Jaundice

Jaundice is a yellowing of the skin and the whites of the eyes that happens when red blood cells break down. It is one of the common features of G6PD deficiency, including in newborns.

Limited evidenceSource: OMIM:300908
Evidence ratingweak
Study designontology_import
Confidence (0-1)0.7
Replicationunreplicated
Supporting sourcesPMID:30295264
Notesplain_language confirmed from PMID:30295264 via curation 2026-06-12.
Last reviewed2026-06-12

Abdominal pain

Belly (abdominal) pain can occur during an acute hemolytic episode, and is more common in children whose episode was set off by fava beans.

Limited evidenceSource: OMIM:300908
Evidence ratingweak
Study designontology_import
Confidence (0-1)0.7
Replicationunreplicated
Supporting sourcesPMID:39956941
Notesplain_language confirmed from PMID:39956941 via curation 2026-06-12.
Last reviewed2026-06-12

Fava bean-induced hemolytic anemia

Favism is the name for an acute episode of red-cell breakdown set off by eating fava beans. It is a well-known cause of sudden hemolytic anemia in people with G6PD deficiency.

Limited evidenceSource: OMIM:300908
Evidence ratingweak
Study designontology_import
Confidence (0-1)0.7
Replicationunreplicated
Supporting sourcesPMID:34964759
Notesplain_language confirmed from PMID:34964759 via curation 2026-06-12.
Last reviewed2026-06-12

Treatment and management

No disease-modifying treatment is established for this condition in the research mapped here. This is a stated, reviewed fact, not a missing piece of this guide.

That does not mean nothing can be done. Supportive and symptomatic care, managing specific symptoms and complications as they arise, can still matter a great deal. What is right for any individual is a conversation for their own care team.

Triggers to avoid

In this condition, certain drugs, foods, or other exposures can set off an acute episode in people who are affected, even when they are otherwise well. The research mapped here describes the agents below. This is information, not a recommendation: what to avoid and what is safe for any individual is a conversation for their own care team.

Fava beans

Eating fava beans is a well-recognized trigger of acute hemolysis in people with G6PD deficiency and is something to avoid.

Reported in the research mapped here as able to provoke: Hemolytic anemia, G6PD deficient (favism) in people with this condition.

Limited evidenceSource: PMID:39956941
The source text this rests on
“…exposure to oxidative stressors, such as drugs or fava bean ingestion, can trigger acute hemolytic episodes…”
An excerpt quoted verbatim from the source named above, shown as recorded. The full sentence is in the linked source.
Evidence ratingweak
Confidence (0-1)0.7
Replicationunreplicated
Notesconfirmed from PMID:39956941 via curation 2026-06-12
Last reviewed2026-06-12

Methylene blue

Methylene blue is a drug that should be avoided in G6PD deficiency because it can worsen red-cell breakdown.

Reported in the research mapped here as able to provoke: Hemolytic anemia, G6PD deficient (favism) in people with this condition.

Limited evidenceSource: PMID:34964759
The source text this rests on
“These patients should not receive methylene blue to avoid worsening hemolysis.”
An excerpt quoted verbatim from the source named above, shown as recorded. The full sentence is in the linked source.
Evidence ratingweak
Confidence (0-1)0.7
Replicationunreplicated
Notesconfirmed from PMID:34964759 via curation 2026-06-12
Last reviewed2026-06-12

Dapsone

Dapsone is a drug used for some infections and immune conditions. It can trigger red-cell breakdown in people with G6PD deficiency and is one to avoid or use only with caution.

Reported in the research mapped here as able to provoke: Hemolytic anemia, G6PD deficient (favism) in people with this condition.

Limited evidenceSource: PMID:42172240
The source text this rests on
“Dapsone may cause haemolysis, particularly in individuals with glucose-6-phosphate dehydrogenase (G6PD) deficiency.”
An excerpt quoted verbatim from the source named above, shown as recorded. The full sentence is in the linked source.
Evidence ratingweak
Confidence (0-1)0.7
Replicationunreplicated
Notesconfirmed from PMID:42172240 via curation 2026-06-12
Last reviewed2026-06-12

Pegloticase

Pegloticase is a drug for severe gout. It can trigger oxidative red-cell breakdown in people with G6PD deficiency, so G6PD status should be checked before it is used.

Reported in the research mapped here as able to provoke: Hemolytic anemia, G6PD deficient (favism) in people with this condition.

Limited evidenceSource: PMID:42017087
The source text this rests on
“Pegloticase is an effective therapy for refractory gout but carries a risk of oxidative hemolysis in patients with glucose-6-phosphate dehydrogenase (G6PD) deficiency.”
An excerpt quoted verbatim from the source named above, shown as recorded. The full sentence is in the linked source.
Evidence ratingweak
Confidence (0-1)0.7
Replicationunreplicated
Notesconfirmed from PMID:42017087 via curation 2026-06-12
Last reviewed2026-06-12

Turn this into questions for your doctor

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How to read the evidence labels

Widely acceptedSpecialists broadly agree on this.
Strong evidenceBacked by solid, repeated research.
Moderate evidenceReasonable evidence, still being confirmed.
Limited evidenceSome evidence, but not yet convincing.
Early evidenceAn early finding that needs more study.
Experts disagreeResearchers actively disagree about this.

Where this comes from

This guide is built from 5 published source(s). Every claim above links back to one of them. Click any source ID to read the original on PubMed.

OMIM:300908 · Orphanet/HPO annotations for Hemolytic anemia, G6PD deficient (favism)
PMID:34964759 · Acute hemolysis and methemoglobinemia secondary to fava beans ingestion in a patient with G6PD deficiency: A case report
PMID:39956941 · Fava Bean- Versus Non-Fava Bean-Induced Acute Hemolytic Crisis in Children With Glucose-6-Phosphate Dehydrogenase Defici
PMID:42017087 · Severe Anemia and Acute Kidney Injury After Pegloticase in a Patient With Glucose-6-Phosphate Dehydrogenase Deficiency.
PMID:42172240 · Dapsone and schistocytes: Thrombotic microangiopathy or not?